Abstract:
Event based communication in a spiking neuron network may be provided. The network may comprise units communicating by spikes via synapses. Responsive to a spike generation, a unit may be configured to update states of outgoing synapses. The spikes may communicate a payload data. The data may comprise one or more bits. The payload may be stored in a buffer of a pre-synaptic unit and be configured to accessed by the post-synaptic unit. Spikes of different payload may cause different actions by the recipient unit. Sensory input spikes may cause postsynaptic response and trigger connection efficacy update. Teaching input may be used to modulate plasticity.
Abstract:
Apparatus and methods for activity based plasticity in a spiking neuron network adapted to process sensory input. In one approach, the plasticity mechanism of a connection may comprise a causal potentiation portion and an anti-causal portion. The anti-causal portion, corresponding to the input into a neuron occurring after the neuron response, may be configured based on the prior activity of the neuron. When the neuron is in low activity state, the connection, when active, may be potentiated by a base amount. When the neuron activity increases due to another input, the efficacy of the connection, if active, may be reduced proportionally to the neuron activity. Such functionality may enable the network to maintain strong, albeit inactive, connections available for use for extended intervals.
Abstract:
Apparatus and methods for learning in response to temporally-proximate features. In one implementation, an image processing apparatus utilizes bi-modal spike timing dependent plasticity in a spiking neuron network. Based on a response by the neuron to a frame of input, the bi-modal plasticity mechanism is used to depress synaptic connections delivering the present input frame and to potentiate synaptic connections delivering previous and/or subsequent frames of input. The depression of near-contemporaneous input prevents the creation of a positive feedback loop and provides a mechanism for network response normalization.
Abstract:
Apparatus and methods for activity based plasticity in a spiking neuron network adapted to process sensory input. In one approach, the plasticity mechanism of a connection may comprise a causal potentiation portion and an anti-causal portion. The anti-causal portion, corresponding to the input into a neuron occurring after the neuron response, may be configured based on the prior activity of the neuron. When the neuron is in low activity state, the connection, when active, may be potentiated by a base amount. When the neuron activity increases due to another input, the efficacy of the connection, if active, may be reduced proportionally to the neuron activity. Such functionality may enable the network to maintain strong, albeit inactive, connections available for use for extended intervals.
Abstract:
Apparatus and methods for event based communication in a spiking neuron network. The network may comprise units communicating by spikes via synapses. The spikes may communicate a payload data. The data may comprise one or more bits. The payload may be stored in a buffer of a pre-synaptic unit and be configured to accessed by the post-synaptic unit. Spikes of different payload may cause different actions by the recipient unit. Sensory input spikes may cause postsynaptic response and trigger connection efficacy update. Teaching input spikes trigger the efficacy update without causing the post-synaptic response.