Abstract:
The input (and output) power of a multi-carrier amplifier can be controlled to allow the amplifier to operate at high RF power levels and still remain within a power rating profile. The amplifier (or amplifiers) power is controlled using an aggregate scaling factor. The aggregate scaling factor is generated from a plurality of amplifier scaling factors. Each amplifier scaling factor is generated based on a comparison of a time-averaged total power and a corresponding threshold.
Abstract:
A method of applying one of several resource distribution algorithms to an incoming call to a communication system. The applied resource distribution algorithm is based on resource parameters obtained from a main scheduler of the communication system. Some of the resource parameters include the type of call, the resource usage and the resource allocation scheme. The resource parameters generally describe the amount of resources that are currently being used by the system and which resources are available to the incoming call. The method of the present invention admits the incoming call to the communication system in accordance with one of the resource distribution allocation schemes. In this manner, the resources of the communication system can be efficiently and fairly distributed to different types of users requesting admission to the communication system.
Abstract:
When an overload condition is determined to exist at a base station in a wireless communications system based on the number of unsuccessful access attempts to access the system relative to the successful access attempts to access the system, the base station broadcasts a message to each mobile terminal within its coverage area that changes at least one access probe-related parameter associated with at least one of: the number, the frequency, and the power intensity of the access probes made by these mobile terminals as they attempt to establish access to the wireless network. By so limiting these access probes in some manner when an overload condition is present, the interference caused on the reverse link by many mobile terminals attempting access is reduced.
Abstract:
A method for controlling access of a subscriber station to a wireless communications system facilitates the maintenance of a performance target of the reverse link and desired geographic coverage of the reverse link. The method for controlling access of a subscriber station to a wireless communications system includes measuring a first performance indicator and a second performance indicator for a reverse link associated with a subscriber station seeking access to a wireless communications system. A blocking threshold value is established based upon the measured second performance indicator and may be adjusted to allow more or less blocking based on any additional metrics that characterize the performance of the current subscriber stations. The decision to grant or deny access of the subscriber station to the wireless communications system is determined based on a comparison of the measured first performance indicator to the established blocking threshold value.
Abstract:
The input (and output) power of a multi-carrier amplifier can be controlled to allow the amplifier to operate at high RF power levels and still remain within a power rating profile. The amplifier (or amplifiers) power is controlled using an aggregate scaling factor. The aggregate scaling factor is generated from a plurality of amplifier scaling factors. Each amplifier scaling factor is generated based on a comparison of a time-averaged total power and a corresponding threshold.
Abstract:
This application discloses a novel process for the preparation of 1,2-substituted 3,4-dioxo-1-cyclobutene compounds, which have utility, for example, in the treatment of CXC chemokine-mediated diseases, and intermediates useful in the synthesis thereof.
Abstract:
This invention relates to compounds of the general formula: in which the variable groups are as defined herein, and to their preparation and use.
Abstract:
Caspase activity and apoptosis are promoted using active, dimeric Smac peptide mimetics of the general formula M1-L-M2, wherein moieties M1 and M2 are monomeric Smac mimetics and L is a covalent linker. Target cancerous or inflammatory cells are contacted with an effective amount of an active, dimeric Smac mimetic, and a resultant increase in apoptosis of the target cells is detected. The contacting step may be effected by administering to a pharmaceutical composition comprising a therapeutically effective amount of The compoundic mimetic, wherein the individual may be subject to concurrent or antecedent radiation or chemotherapy for treatment of a neoproliferative pathology.
Abstract:
Caspase activity and apoptosis are promoted using active, dimeric Smac peptide mimetics of the general formula M1-L-M2, wherein moieties M1 and M2 are monomeric Smac mimetics and L is a covalent linker. Target cancerous or inflammatory cells are contacted with an effective amount of an active, dimeric Smac mimetic, and a resultant increase in apoptosis of the target cells is detected. The contacting step may be effected by administering to a pharmaceutical composition comprising a therapeutically effective amount of The compoundic mimetic, wherein the individual may be subject to concurrent or antecedent radiation or chemotherapy for treatment of a neoproliferative pathology.