CULTURED EXTREMOPHILIC ALGAE SPECIES NATIVE TO NEW MEXICO
    4.
    发明申请
    CULTURED EXTREMOPHILIC ALGAE SPECIES NATIVE TO NEW MEXICO 审中-公开
    新墨西哥州的文化的极端的阿尔及利亚种

    公开(公告)号:US20130164322A1

    公开(公告)日:2013-06-27

    申请号:US13723687

    申请日:2012-12-21

    CPC classification number: C12N1/12 C12R1/89

    Abstract: Provided herein is an extremophile green alga designated as Scenedesmus species Novo, from Jemez warm water springs, New Mexico. Sequencing 18S rDNA confirmed the alga as a new species. It is capable of producing high levels of microalgal biomass in wastewater under harsh ambient climatic conditions, and of yielding high levels of lipids and carotenes. Cultures in TAP medium at 24±1° C. at continuous light (132-148 μmol photons m−2s−1) attained peak biomass levels 27.4×106 cells ml−1, 49.11 μg ml−1 chlorophyll α, 24.93 μg ml−1 carotene on the seventh day and a division rate of 0.54 day−1. High levels of biomass were sustained in sterilized and unsterilized municipal wastewater, enriched with 1% TAP nutrients or unenriched. The microalga is useful in the production of biofuels, fertilizers, dietary nutrients, pharmaceuticals, polymers, biofilters to remove nutrients and other pollutants from wastewaters, in space technology, and laboratory research systems.

    Abstract translation: 这里提供的是一种极端微生物绿藻,被称为Scenedesmus物种Novo,来自Jemez温泉泉,新墨西哥州。 测序18S rDNA确认藻类为新物种。 它能够在恶劣的环境气候条件下在废水中产生高水平的微藻生物质,并产生高水平的脂质和胡萝卜素。 在TAP培养基中在24±1℃的连续光培养(132-148μmol光子m-2s-1)达到峰值生物量27.4×10 6个细胞ml-1,49.11毫升ml-1叶绿素α,24.93毫升ml- 1胡萝卜素,分化率为0.54天-1。 消毒和未灭菌的城市废水中维持高水平的生物量,富含1%TAP营养或未富集。 微藻可用于生物燃料,肥料,膳食营养素,药物,聚合物,生物过滤器,以从废水,空间技术和实验室研究系统中去除营养物质和其他污染物。

    Peptide inhibitors of LFA-1/ICAM-1 interaction
    5.
    发明申请
    Peptide inhibitors of LFA-1/ICAM-1 interaction 有权
    LFA-1 / ICAM-1相互作用的肽抑制剂

    公开(公告)号:US20010034326A1

    公开(公告)日:2001-10-25

    申请号:US09760599

    申请日:2001-01-16

    CPC classification number: C07K7/06 A61K38/12 C07K7/64

    Abstract: Cyclic peptides inhibit LFA-1 interaction with ICAM-1 and are useful in treatment of hematopoietic neoplasms and in adjunct therapy in prevention of retinoic acid syndrome and diseases involving emigration of leukocytes into organ tissue.

    Abstract translation: 循环肽抑制LFA-1与ICAM-1的相互作用,可用于治疗造血赘生物和辅助治疗预防视黄酸综合征和涉及白细胞移入器官组织的疾病。

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